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The labels are being revised to include a warning to avoid use in patients with congenital long QT syndrome because these patients are at particular risk for Torsade. The Zofran (ondansetron) drug labels already contain information about the potential for QT prolongation. Additional label changes may result after the additional information has been reviewed. The results from this study are expected to be available in the summer of 2012. The manufacturer of Zofran (GlaxoSmithKline) is being required to conduct a thorough QT study to assess the potential for the drug to prolong the QT interval.
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Patients at particular risk for developing Torsade include those with underlying heart conditions, such as congenital long QT syndrome, those who are predisposed to low levels of potassium and magnesium in the blood, and those taking other medications that lead to QT prolongation.įDA has reviewed all available information and is making interim changes to the drug labels. Also available as an injection for intravenous use (2 mg/mL).Ĭhanges in the electrical activity of the heart (prolongation of the QT interval of the electrocardiogram ) - see Data Summary below - can lead to an abnormal and potentially fatal heart rhythm (including Torsade de Pointes). Available as 4 mg and 8 mg tablets, 4 mg and 8 mg orally disintegrating tablets, and oral solution (4 mg/5 mL).Works by blocking the action of serotonin, a natural substance that may cause nausea and vomiting. Is in a class of medications called 5-HT3 receptor antagonists.Used to prevent nausea and vomiting caused by cancer chemotherapy, radiation therapy and surgery.Has my activity level been and is it still enough to cause mild dilation? Your advice would be much appreciated.Facts about Zofran (ondansetron and ondansetron hydrochloride) No other action to be taken … I am trying to understand what the probability of early DCM is in my situation. However, … I am to have a repeat MRI in a year’s time. He considered it unlikely it was dilated cardiomyopathy. … MRI showed mild to moderate dilation to both ventricles … The cardiologist explained that in his opinion the dilation was likely due to my size (I am tall) and my activity level. During sleep my HBPM was 35 at its lowest – an exercise test, which showed the PVCs particularly well. I was referred to a cardiologist and the following tests were done: – an ultrasound, which showed my heart was structurally ok- 24 hr monitor, which showed 231 multifocal PVCs and 200 sinus arrhythmia, total heartbeats ~79,900. Since recently reducing caffeine and starting with extra magnesium the symptoms have dramatically reduced …. My resting pulse is low, between 40-50 bpm …. About two months ago the PVCs flared up again, this time related to activity and exercise …. The test showed I had a high level of fitness … and that my heart performed better than expected for my age and gender under stress …. Last year in Jan 2012 I had an extensive health check and part of the check was fitness. Since the children I have remained active, only … frequency is now not as great …. Prior to my children being born I was exercising 5 times per week … at quite high intensity …. Since then I started exercising a lot more and lost a significant amount of weight (I was 143lbs at my lowest).
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At the time it was determined (after an ultrasound, 24 hr monitor and stress test) that these were symptoms of a healthy heart and benign, mainly originating from the right ventricle …. About 10 years ago I was investigated for premature ventricular contractions. I am a 34 year old female, 1.75m (roughly 5’9″), currently 76kg (167lbs). I would be very interested in and would value your opinion of my situation, which I detail below. Question: Can you help me determine my probability for dilated cardiomyopathy (DCM)?